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Quigg, Richard
One or more keywords matched the following items that are connected to Quigg, Richard
Item TypeName
Concept Mice, Inbred MRL lpr
Academic Article Transgenic expression of a soluble complement inhibitor protects against renal disease and promotes survival in MRL/lpr mice.
Academic Article Excessive matrix accumulation in the kidneys of MRL/lpr lupus mice is dependent on complement activation.
Academic Article Administration of the soluble complement inhibitor, Crry-Ig, reduces inflammation and aquaporin 4 expression in lupus cerebritis.
Academic Article Administration of a soluble recombinant complement C3 inhibitor protects against renal disease in MRL/lpr mice.
Academic Article C5a promotes development of experimental lupus nephritis which can be blocked with a specific receptor antagonist.
Academic Article Complement-dependent apoptosis and inflammatory gene changes in murine lupus cerebritis.
Academic Article CR1/CR2 deficiency alters IgG3 autoantibody production and IgA glomerular deposition in the MRL/lpr model of SLE.
Academic Article Absence of functional alternative complement pathway alleviates lupus cerebritis.
Academic Article Signaling through up-regulated C3a receptor is key to the development of experimental lupus nephritis.
Academic Article Inhibition of C5a receptor alleviates experimental CNS lupus.
Academic Article C5a alters blood-brain barrier integrity in experimental lupus.
Academic Article C3aR inhibition reduces neurodegeneration in experimental lupus.
Academic Article Alteration in kidney glucose and amino acids are implicated in renal pathology in MRL/lpr mice.
Academic Article MRL/lpr mice have alterations in brain metabolism as shown with [1H-13C] NMR spectroscopy.
Academic Article Complement factor H deficiency accelerates development of lupus nephritis.
Academic Article C5a induces caspase-dependent apoptosis in brain vascular endothelial cells in experimental lupus.
Academic Article Curcumin aggravates CNS pathology in experimental systemic lupus erythematosus.
Grant Pathogenic role of the complement system in murine lupus
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  • Mice Inbred MRL lpr